pg_0001

Medical data is for informational purposes only. You should always consult your family physician, or one of our referral physicians prior to treatment.

Supplement to

The Art of Getting Well

Molybdenum for Candida albicans

Patients and Other Problems

Sources are given in references.

Authors of contributions\quotations are alphabetically arranged;

major author, if any, is underlined.

Dr. George Miroff, Dr. Richard Mowles, Jon B.Pangborn,

William H. Philpott, M.D./Responsible editor/writer Walter H.

Schmitt, Jr., D.C.

Foundation for the Eradication of Rheumatoid Disease

AKA The Arthritis Trust of America

7376 Walker Road, Fairview, Tn 37062

Published originally in The Digest of Chiropractic Economics, 31:4, January-

February, 1991, pp. 56-63, 29229 Six Mile Road, Livonia, Michigan 48152-3661, Phone

(313) 427-5720, and is reprinted as it appeared with permission of the author and

Chiropractic News Publishing Company, Inc., courtesy of Keith A. Tosolt, Managing

Editor.

ABSTRACT: The essential trace element, molybdenum, (pro-

nounced mo-lib'de-num) is discussed in relation to its various

metabolic pathways. Diagnostic approaches for molybdenum in-

clude applied kinesiological procedures based on strong muscles

weakening when a patient sniffs aldehydes, ammonia, or Clorox, or

tastes sulfur-containing amino acids. Other patterns indicating a

need for molybdenum are the same as would be seen in a need for iron

and/or excess of copper. Each of these metabolic pathways are

shown to be important in the problems of the Candida albicans

patient, as well as other patients. Protocols for supplementation and

natural sources of molybdenum are given.

INTRODUCTION: Molybdenum is an essential trace element

in human nutrition which is understood about as well as it is

pronounced. In fact, there is no laboratory testing which has been

standardized for the evaluation of molybdenum. Although it has

been measured in both blood and hair, the normal values for these

tests have yet to be established, and although it is accepted as an

essential nutrient for humans, there has yet to be a recommended

daily allowance or minimum daily requirement officially estab-

lished. However, its importance in numerous patients, including

those with Candida albicans allergy, has been paramount.

Molybdenum has been studied both directly in the blood and

hair

, indirectly by looking at other metabolites which relate to the

presence of molybdenum

2,3

and by applied kinesiological (A.K.)

analysis by Dr. Richard Mowles

Molybdenum is necessary for the function of at least three

important enzymes in the body: 1) aldehyde oxidase for our bodies'

handling of aldehydes it produces and those encountered in the

environment; 2) xanthine oxidase for the conversion of purines into

uric acid; and 3) sulfite oxidase for the conversion of irritating

sulfites into harmless sulfates. In addition, molybdenum is found in

many biological processes in conjunction with iron and is found to

cause a response in AK indicators similar to that of iron. Also,

molybdenum is an antagonist to copper and vice versa. Considering

all of the above factors has led to our understanding of how

molybdenum is usually a necessary adjunct to the treatment of

Candida albicans allergy patients and has speeded the recovery of

most of these patients even above and beyond the effective natural

procedures which were described in a previous paper by Mowles and

this author

MOLYBDENUM AND ALDEHYDES: Chemical aldehydes

are best known as fragrances. The body also produces various

aldehydes as part of its normal metabolic pathways. One pathway in

the metabolism of the essential amino acid, threonine, is its

conversion into acetaldehyde and then on into acetic acid for

eventual production of acetyl coenzyme A.

Acetaldehyde is a particularly toxic substance which, in

addition to being produced from threonine and ethanol, is a product

of the metabolism (i.e. fermentation) of carbohydrate in yeast --

hence, the Candida connection. Acetaldehyde is thought to be the

major source of tissue damage in alcoholics rather than ethanol

itself. The conversion of acetaldehyde into acetic acid is shown in

Figure 2. Note that this reaction requires NAD (niacinamide), and

aldehyde oxidase is dependent on FAD (riboflavin), iron (Fe), and

Molybdenum (Mo).

Candida albicans patients and any other patients who com-

plain of sensitivities to various fragrances and airborne odors will

be found to have a problem with an olfactory challenge with an

aldehyde and will be found to be in need of one or more of the

nutrients associated with the metabolism of aldehydes, that is

niacinamide, riboflavin, iron and/or molybdenum. Mowles

had

patients sniff a dilute source of formaldehyde and observed the

results in muscle testing of patients. In 15 patient trials with

weakening on smelling formaldehyde, 14 were found to strengthen

on molybdenum. Our clinical procedure paralleled that of Mowles,

although we used different sources of aldehydes.

Our original investigation involved using nail polish remover

(Cutex

brand) as a source of acetone. We were attempting to use

an olfactory challenge for ketones since transketolase enzyme is

vitamin B

(thiamine) dependent. We had performed the functional

blood test for red blood cell transketolase on a number of patients

and found some of them to show a need for B

. Knowing that nail

polish remover is primarily acetone, we attempted olfactory chal-

lenging to observe the results. Eventually, we switched to using

pure acetone for olfactory challenging because of the sporadic

results we observed from using the nail polish remover.

A close look at the contents of the Cutex bottle revealed that

besides acetone, a fragrance and a food color had been added. It is

useful here to mention when testing with sniffing acetone, a

strengthening of weak muscles or a weakening of strong muscles

has been demostrated to be associated with a need for vitamin B

The proper B

tablet, either high synthetic doses or low, natural

source, or occasionally both, when insalivated, will block the

weakening response to sniffing acetone in those patients who show

it, and will likewise mimic the strengthening response in that group

of patients.

Although we still occasionally use the nail polish remover as

a screening test for acetone and B

involvement, we now know that

many of our original sporadic observations were due to the pres-

ence of a fragrance (i.e., an aldehyde) in the product. We obtained

a source of benzaldehyde, which is the smell of almonds and quite