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Medical data is for informational purposes only. You should always consult your family physician, or one of our referral physicians prior to treatment.

Table of Arthritides' Symptoms and Their Classifications*

Table I: Collagen Tissue Diseases (Rheumatoid Diseases), and Associated Rheumatic Disease Conditions

Collagen Tissue is the main organic constituent of connective tissue and of the organic substance of bones. As such, it is pervasive

throughout the human body.

Clinical Name of Disease

Symptoms , Onset and Body Locations

Acute Cholecystitis

See Table II.

Acute Meningitis

See Headache, Table I and Table II.

Acute Pancreatitis

See Pancreatitis, Table I.

Adenoid Hyperplasia

Adenoid Hyperplasia is enlargement of adenoidal tissue due to lymphoid excessive prolifera-

tion of normal cells in a normal arrangement (hyperplasia). This condition occurs in children

and may be physiologic or secondary to infection or allergy. Obstruction of the eustachian tubes

may result in recurrent acute, chronic, or secretory (serous) otitis media. Obstruction of the

passageway between the nose and pharynx (choana) may cause mouth breathing, a hyponasal

voice and pus forming or running in the nostril (purulent rhinorrhea).

Allergic Interstitial Penumonitis

See Hypersensitivity Penumonitis, Table I.

Alopecia

Partial or complete loss of hair resulting from genetic factors, aging, or local or systemic disease,

such as seborrheic dermatitis and Psoriasis. See Psoriasis, Table I.

ALS

See Amyotrophic Lateral Sclerosis, Table I.

Amyloid Arthropathy

See Amyloidosis, Table I.

Amyloidosis

Amyloid resembles starch, and is a white insoluble protein substance found as an abnormal

deposit in various organs. Homeogenous, translucent and colorless, it consists of protein-

carbohydrate fiberous or filamentous (fibrillar) amyloid in amounts sufficient to impair normal

function of the tissues or organs. The appearance of Amyloidosis may or may not be associ-

ated with a specific disease. It can be found associated with chronic diseases such as tuberculosis,

Alzheimer's disease, dilatation of the broncchial tubes (bronchiectasis), inflammation of the

bone and marrow (osteomyelitis), leprosy, marrow plasma cell tumors (multiple myeloma),

lymph system cancer (Hodgkin's disease) and other tumors, or inflammatory Rheumatoid

Arthritis . In primary Amyloidosis -- without associated disease -- the heart, lung, skin,

tongue, thyroid gland and intestinal tract may be involved. Localized amyloid "tumors" may be

found in the respiratory tract or other sites. The liver, spleen, kidney and vasuclar system are

frequently involved. In secondary Amyloidosis -- with associated disease -- there is a predilec-

tion for the spleen, liver, kidney, adrenal glands and lymph nodes. However, no organ is spared

and vascular involvement may be widespread. The liver and spleen are enlarged, firm and

rubbery. Kidneys are usually enlarged. Sections of the spleen show large, translucent, waxy areas.

Amyloid associated with certain tumors (multiple myeloma) may be widespread and

may show unique sites of involvement, or the Amyloid may be a local occurrence associated with

some malignancies.. Symptoms are non-specific and will often originate in the organ or system

affected by the Amyloid tissues.There are, therefore, different, distinct pathologic signs and

symptoms depending upon the organ or system affected. Some forms may resemble cardiac

failure, or nerve dysfunction (peripheral neuropathy), skin disease, or Hashimoto's

or Reidel's Struma. Amyloid Arthropathy may mimic Rheumatoid Arthritis in some

cases of multiple myeloma (Cancer). Secondary Amyloidosis recovery depends on success-

ful treatment of the underlying disease. If successful, then, Amyloidosis can be arrested.

Amyotrophic Lateral Sclerosis

See Muscular Atrophies, Table I.

Anaphylactoid Purpura

See Allergic Purpura, Table II.

Anemia of Chronic Blood Loss

See Anemia, Pernicious, Table I. Also see Splenomegaly, Table I.

Anemia, Pernicious

A gradually increasing increase in size of red corpuscles (macrocytic) and lowered color index of

blood (hypochromic) anemia with usually deficient supply of hydrochloric acid (achlorhy-

dria) and a deficiency of a specific factor with enzyme-like properties present in normal gastric

juice, called "intrinsic factor," or "Castle's Intrinsic." This factor interacts with food to produce

the antipernicious anemia principle which is required to transport B

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across the intestinal

mucosa. Anemia develops in most patients insidiously and progressively as large liver (hepatic)

stores are depleted. Gasterectomy, Chronic Atrophic Gastritis, Table I, and Myxedema,

Table I, may also cause deficiency. Competition for B

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may also occur from invasive organisms.

Absorptive sites in the Ileium may be destroyed by inflammatory Regional Enteritis, surgical

resection, or because the producing cells are congenitally absent. There are other causes, such

as malabsorption syndromes, chronic pancreatitis and use of various drugs. Enlargement of the

spleen (Splenomegaly, Table I) and liver (Heptomegaly, Table I) may sometimes

be seen. Various gastrointestinal manifestations may be present, including loss of appetite

(anorexia), intermittant constipation and diarrhea, poorly localized abdominal pain, a sensation

of burning of the tongue (glossitis), and weight loss. A transient burning or prickling sensation

(paresthesia) may be felt in the upper extremities, or other peripheral nerve sensations (periph-

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